Critical POU domain residues confer Oct4 uniqueness in somatic cell reprogramming

Oct4 redox sensitivity potentiates iPSC reprogramming

Evolutionary Origin of Vertebrate OCT4/POU5 Functions in Supporting Pluripotency

Proteomic Analysis of Mouse Oocytes Identifies PRMT7 as a Reprogramming Factor that Replaces SOX2 in the Induction of Pluripotent Stem Cells

PDF] The Oct4 protein: more than a magic stemness marker.

Oct4 -- The Gatekeeper of Pluripotency of Embryonic Stem Cells

Reprogramming enriches for somatic cell clones with small-scale mutations in cancer-associated genes - ScienceDirect

Regulation of Oct4 in stem cells and neural crest cells - Patel - 2022 - Birth Defects Research - Wiley Online Library

Site-specific Disruption of the Oct4/Sox2 Protein Interaction Reveals Coordinated Mesendodermal Differentiation and the Epithelial-Mesenchymal Transition* - Journal of Biological Chemistry

Critical POU domain residues confer Oct4 uniqueness in somatic cell reprogramming

Evolutionary Origin of Vertebrate OCT4/POU5 Functions in Supporting Pluripotency

Enhancing Sox/Oct cooperativity induces higher-grade developmental reset

Changing POU dimerization preferences converts Oct6 into a pluripotency inducer

Critical POU domain residues confer Oct4 uniqueness in somatic cell reprogramming

Oct4 induced iPSCs to express pluripotency markers and resemble ESCs.

Changing POU dimerization preferences converts Oct6 into a pluripotency inducer