Critical POU domain residues confer Oct4 uniqueness in somatic cell reprogramming
Por um escritor misterioso
Last updated 20 outubro 2024
Oct4 redox sensitivity potentiates iPSC reprogramming
Evolutionary Origin of Vertebrate OCT4/POU5 Functions in Supporting Pluripotency
Proteomic Analysis of Mouse Oocytes Identifies PRMT7 as a Reprogramming Factor that Replaces SOX2 in the Induction of Pluripotent Stem Cells
PDF] The Oct4 protein: more than a magic stemness marker.
Oct4 -- The Gatekeeper of Pluripotency of Embryonic Stem Cells
Reprogramming enriches for somatic cell clones with small-scale mutations in cancer-associated genes - ScienceDirect
Regulation of Oct4 in stem cells and neural crest cells - Patel - 2022 - Birth Defects Research - Wiley Online Library
Site-specific Disruption of the Oct4/Sox2 Protein Interaction Reveals Coordinated Mesendodermal Differentiation and the Epithelial-Mesenchymal Transition* - Journal of Biological Chemistry
Critical POU domain residues confer Oct4 uniqueness in somatic cell reprogramming
Evolutionary Origin of Vertebrate OCT4/POU5 Functions in Supporting Pluripotency
Enhancing Sox/Oct cooperativity induces higher-grade developmental reset
Changing POU dimerization preferences converts Oct6 into a pluripotency inducer
Critical POU domain residues confer Oct4 uniqueness in somatic cell reprogramming
Oct4 induced iPSCs to express pluripotency markers and resemble ESCs.
Changing POU dimerization preferences converts Oct6 into a pluripotency inducer
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