Mitochondrial reactive oxygen is critical for IL-12/IL-18-induced IFN
Por um escritor misterioso
Last updated 16 janeiro 2025
Frontiers Reactive Oxygen Species Interact With NLRP3 Inflammasomes and Are Involved in the Inflammation of Sepsis: From Mechanism to Treatment of Progression
Doxorubicin-Induced Oxidative Stress and Endothelial Dysfunction in Conduit Arteries Is Prevented by Mitochondrial-Specific Antioxidant Treatment
Mitochondrial reactive oxygen is critical for IL-12/IL-18-induced IFN-γ production by CD4+ T cells and is regulated by Fas/FasL signaling
Mitochondrial Functions in Infection and Immunity: Trends in Cell Biology
Mitochondria as a centrally positioned hub in the innate immune response - ScienceDirect
Frontiers Mitochondrial Reactive Oxygen Species Are Essential for the Development of Psoriatic Inflammation
IJMS, Free Full-Text
Interferon Gamma Induces Reversible Metabolic Reprogramming of M1 Macrophages to Sustain Cell Viability and Pro-Inflammatory Activity - eBioMedicine
Mitochondrial respiration contributes to the interferon gamma response in antigen-presenting cells
Mitochondrial reactive oxygen is critical for IL-12/IL-18-induced IFN-γ production by CD4+ T cells and is regulated by Fas/FasL signaling
Journal of Cellular Physiology, Cell Biology Journal
Mitochondrial reactive oxygen is critical for IL-12/IL-18-induced IFN-γ production by CD4+ T cells and is regulated by Fas/FasL signaling
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