CRISPR-Cas9 correction of OPA1 c.1334G>A: p.R445H restores mitochondrial homeostasis in dominant optic atrophy patient-derived iPSCs - ScienceDirect
Por um escritor misterioso
Last updated 31 março 2025


Understanding the molecular basis and pathogenesis of hereditary optic neuropathies: towards improved diagnosis and management - ScienceDirect

OPA1 regulation of mitochondrial dynamics in skeletal and cardiac muscle - ScienceDirect

PDF) CRISPR/Cas9 correction of OPA1 c.1334G>A: p.R445H restores mitochondrial homeostasis in Dominant Optic Atrophy patient-derived iPSCs

CRISPR-Cas9 correction of OPA1 c.1334G>A: p.R445H restores mitochondrial homeostasis in dominant optic atrophy patient-derived iPSCs: Molecular Therapy - Nucleic Acids

PDF) CRISPR/Cas9 correction of OPA1 c.1334G>A: p.R445H restores mitochondrial homeostasis in Dominant Optic Atrophy patient-derived iPSCs

PDF) CRISPR/Cas9 correction of OPA1 c.1334G>A: p.R445H restores mitochondrial homeostasis in Dominant Optic Atrophy patient-derived iPSCs

A Genome-wide CRISPR Death Screen Identifies Genes Essential for Oxidative Phosphorylation - ScienceDirect

Functional genomics and the future of iPSCs in disease modeling - ScienceDirect

Co-opting regulation bypass repair as a gene-correction strategy for monogenic diseases: Molecular Therapy

PDF) CRISPR/Cas9 correction of OPA1 c.1334G>A: p.R445H restores mitochondrial homeostasis in Dominant Optic Atrophy patient-derived iPSCs

Dominant optic atrophy, OPA1, and mitochondrial quality control: understanding mitochondrial network dynamics, Molecular Neurodegeneration
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