Imbalance of Neuregulin1-ErbB2/3 signaling underlies altered myelin homeostasis in models of Charcot-Marie-Tooth disease type 4H
Por um escritor misterioso
Last updated 06 janeiro 2025
bioRxiv - the preprint server for biology, operated by Cold Spring Harbor Laboratory, a research and educational institution
PDF] Myelin is dependent on the Charcot–Marie–Tooth Type 4H
Enhanced AKT activity and unaltered expression levels of Dlg1 and
Focal hypermyelination in PTEN mutants.A.Westernblot analysis of
Imbalance of Neuregulin1-ErbB2/3 signaling underlies altered
Imbalance of NRG1-ERBB2/3 signalling underlies altered myelination
Schwann cells, but not Oligodendrocytes, Depend Strictly on
Charcot-Marie-Tooth Disease: CMT2, CMT4, And Others
Proteostasis plays an important role in demyelinating Charcot
ErbB receptor-mediated signaling in regulation of myelination
Ablation of Frabin/Fgd4 reduces activation of the RhoGTPase Cdc42
MFN2 R94Q induces ER stress both in vitro and in vivo. (A
Clinical genetics of Charcot–Marie–Tooth disease
Clinical implications of genetic advances in Charcot–Marie–Tooth
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