SARS-CoV-2 evades natural killer cell cytotoxic responses

In a recent study posted to the bioRxiv* preprint server, researchers at Stanford University School of Medicine revealed the mechanism via severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) escapes natural killer (NK) cell cytotoxic reactions.

Pyroptotic cell death in SARS-CoV-2 infection: revealing its roles during the immunopathogenesis of COVID-19

Immune system evasion – the HIV-infected cell

HCMV-secreted glycoprotein gpUL4 inhibits TRAIL-mediated apoptosis and NK cell activation

Frontiers Covid-19: Perspectives on Innate Immune Evasion

Pathways towards human immunodeficiency virus elimination - eBioMedicine

The Development of Natural Killer Cells for Solid Tumor Treatment

Molecular mechanisms involved in pathogenicity of SARS-CoV-2: Immune evasion and implications for therapeutic strategies - ScienceDirect

Herpesvirus Evasion of Natural Killer Cells

Research, Catherine Blish Lab

SARS-CoV-2 host-shutoff impacts innate NK cell functions, but antibody-dependent NK activity is strongly activated through non-spike antibodies