Non-canonical NFκB activation promotes chemokine expression in podocytes
Por um escritor misterioso
Last updated 11 abril 2025


MAP3K kinases and kidney injury

Podocyte derived TNF‐α mediates monocyte differentiation and contributes to glomerular injury - Nishad - 2022 - The FASEB Journal - Wiley Online Library

TWEAK, a multifunctional cytokine in kidney injury - ScienceDirect

EGF-Receptor–Dependent TLR7 Signaling in Macrophages Promotes Glomerular Injury in Crescentic Glomerulonephritis - Laboratory Investigation

IJMS, Free Full-Text

The non-canonical NF-κB pathway in immunity and inflammation

IJMS, Free Full-Text

Inflammasome activation in podocytes: a new mechanism of glomerular diseases

NF-κB in inflammation and renal diseases, Cell & Bioscience

IJMS, Free Full-Text

Cells, Free Full-Text

JCI Insight - A20 and the noncanonical NF-κB pathway are key regulators of neutrophil recruitment during fetal ontogeny

Non-canonical NF-κB signaling in rheumatoid arthritis: Dr Jekyll and Mr Hyde?, Arthritis Research & Therapy

Pristimerin effectively inhibits the malignant phenotypes of uveal melanoma cells by targeting NF‑κB pathway

PDF) Non-canonical NFκB activation promotes chemokine expression in podocytes
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