Adenylate kinase 9 is essential for sperm function and male
Por um escritor misterioso
Last updated 14 abril 2025


The association of ODF4 with AK1 and AK2 in mice is essential for fertility through its contribution to flagellar shape

Pedigree with AK7 mutation and Papanicolaou staining of the proband's

Deficiency in AK9 causes asthenozoospermia and male infertility by destabilising sperm nucleotide homeostasis - ScienceDirect

IJMS, Free Full-Text

Towards Post-Meiotic Sperm Production: Genetic Insight into Human Infertility from Mouse Models

Adenylate Kinases 1 and 2 Are Part of the Accessory Structures in the Mouse Sperm Flagellum1

Phosphoproteomic and Functional Analyses Reveal Sperm-specific Protein Changes Downstream of Kappa Opioid Receptor in Human Spermatozoa - ScienceDirect

AMPing up and down-integration cellular AMP signals by adenylate kinase.

Deficiency in AK9 causes asthenozoospermia and male infertility by destabilising sperm nucleotide homeostasis - eBioMedicine

PDF) Adenylate Kinase and AMP Signaling Networks: Metabolic Monitoring, Signal Communication and Body Energy Sensing

Adenylate kinase 9 is essential for sperm function and male fertility in mammals

Frontiers Roles of Nitric Oxide in the Regulation of Reproduction: A Review

GIV/Girdin, a non-receptor modulator for Gαi/s, regulates spatiotemporal signaling during sperm capacitation and is required for male fertility

GIV/Girdin, a non-receptor modulator for Gαi/s, regulates spatiotemporal signaling during sperm capacitation and is required for male fertility
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